Chondroplasia (ACH) is a common form of chondrodysplasia, also known as chondrodystrophia fetalis and chondrodystrophic dwarfism. The pathogenesis of ACH is closely related to point mutations in the transmembrane region of the fibroblast growth factor receptor 3 (FGFR3) gene, and 80% to 90% of cases are disseminated and are de novo mutations. Spinal deformities in ACH patients often lead to serious outcomes such as urinary impairment, claudication and even paraplegia. This article focuses on a review of spinal deformities and treatment due to ACH. 1, clinical manifestations: the incidence of ACH is low, 1/15000 to 1/40000. the main clinical manifestations are deformities of the skeletal system. Ain et al. pointed out that 95% of patients with ACH from birth to 6 months of age can have 15-25 thoracolumbar kyphosis; from 6 to 18 months of age, the kyphosis can increase to 60-70 due to sitting up; the kyphosis is mainly concentrated in the range of T10 to L4. After the patient walks independently, the kyphosis can be relieved by itself in some patients with kyphosis. The incidence of kyphosis in patients aged 1 to 2 years is 87%; it decreases to 39% at the age of 2 to 5 years; without therapeutic intervention, permanent thoracolumbar kyphosis occurs in about 11% of patients. Permanent thoracolumbar kyphosis can develop progressively and cause increased anterior lumbar convexity and spinal cord compression at the apex of the kyphosis, resulting in neurological symptoms such as abnormal sensation, back pain, and urinary incontinence. It is also noted that the above symptoms are mostly seen in patients aged 15 to 30 years. Schkrohowsky et al. noted that about 20% to 50% of patients with ACH have symptoms of spinal stenosis, and the onset of symptoms is mostly in patients aged 30 to 40 years. Other clinical manifestations include characteristic short-limbed dwarfism, in which patients are short in stature and have short limbs. According to statistics, by maturity, the average height of ACH patients is 131 (5.6) cm for males and 124 (5.9) cm for females; the fingers are thick and short, and are separated, often in groups of 4 or 5 fingers, 2 or 3 fingers, and the thumb, resembling a “trident”; the elbow joints cannot be straightened, and the legs are “O” shaped. “O” shape. At birth, the head can be found to be large and small, with a collapsed frontal nose, nostrils facing forward and a protruding lower jaw. Some patients may have mild hydrocephalus. The trunk development is nearly normal, the ribs are short, the sternum is short and wide, the thorax is flat, the abdomen is convex, and the hips are posterior. If the lesions of the skull and spine are mild, the intellectual and physical development is generally good and the sexual function is normal. 2.Imaging: Spine: The main X-ray changes of spinal deformity in ACH patients include small vertebrae with short pedicles and narrow lumbar pedicle spacing. In lumbar spinal stenosis, the transverse diameter of the lumbar spinal canal (i.e., pedicle spacing) gradually becomes smaller from L1 to L5, and the pedicle spacing is L5/L1.