Intraventricular hemorrhage is more common in acute hemorrhagic cerebrovascular disease, with an incidence of 30% to 38%. One of the most common causes of morbidity is hypertensive cerebral hemorrhage. It is easy to cause ventricular casts. The mortality rate of traditional treatment is as high as 75%~100%. I can since February 2001 to June 2005, a total of 29 cases of ventricular casts were admitted, after the use of comprehensive treatment measures, achieved better results. 1, clinical information 1.1 general information male 17 cases, female 12 cases. Age 41~80 years old, average 58 years old. All were confirmed by cranial CT. All had a history of hypertension. At the time of admission, the GCS score was 3-5 points in 16 cases, 6-8 points in 13 cases; 1 case of double pupil dilation, 3 cases of one-side pupil dilation; 20 cases of hemiparesis; all had cervical ankylosis; the onset of the disease to the hospital time was 1h~17h, an average of 4h. There was no sighing respiratory appearances. 1.2 Imaging examination, cranial CT showed that the primary hemorrhage site was in the basal ganglia and the internal capsule in 18 cases, the thalamus in 10 cases, and a simple intracerebral hematoma in 1 case; among them, the hemorrhage volume of intracerebral hematoma ranged from 8 to 26 ml, with an average of 16 ml; the midline shift was less than 1 cm; and hemorrhage involved the bilateral ventricles and the third and fourth ventricles, and the formation of casts. Acute obstructive hydrocephalus was present in 9 cases. 14 cases were combined with old cerebral infarction. 1.3 Treatment Immediately after admission, dehydration to reduce intracranial pressure, application of hemostatic drugs and preoperative preparation, under local anesthesia, percutaneous conical cranial lateral ventricle double frontal horn puncture to extract the stale blood, physiological saline flushing and continuous external drainage tube (Weihai ventricular drain FC-II type), the drain tube in the 1-2 days of the drainage in the low position, and later elevated to the ear screen on the 10cm ~ 15cm. 6 hours after the tube was placed to start the bilaterals and lateral ventricles each injected with urokinase. The lateral ventricles were each injected with 10,000 units of urokinase plus 2 ml of saline, to intraventricular pressure is generally high or low clamping tube 2h after the opening of continuous drainage, twice a day, continuous use of 3 days ~ 4 days. Lumbar puncture was started on the fifth day, once a day for three consecutive days, and then every other day until the cerebrospinal fluid was yellowish or clear. The ventricular drain was removed on the seventh to tenth day. Drainage was performed for an average of eight days. Other comprehensive treatments included tracheotomy, control of blood pressure, prevention of hemorrhage, physical hypothermia, Nimotropic and prevention of emergency ulcers and infections. 2.Results The review CT of this group showed that the cerebral ventricle was patent, and no intracranial infection occurred. Six cases of hydrocephalus were treated with ventriculo-peritoneal shunt. In this group, 5 cases (17.3%) fully resumed their daily life, 10 cases (34.5%) partially resumed their daily life, 9 cases (31.0%) needed care or nursing, and 5 cases (17.3%) died after 6-month postoperative follow-up. 3, DISCUSSION The mortality rate of ventricular hemorrhage can be more than 80%, whether it is direct surgery, internal conservative treatment, or simple external ventricular drainage [4]. This is due not only to the occupying compression of the hematoma, midline shift, and brainstem damage, but also to the obstruction of the ventricular system by the accumulation of blood in the ventricles. Although when intracerebral hemorrhage breaks into the ventricles, the damage caused by hemorrhage and the increase in intracranial pressure are relatively mild due to the “internal decompression” caused by the hematoma breaking into the ventricles, the blockage of the ventricular system by the blood clot or the compression of the aqueduct leading to acute obstructive cerebrospinal fluid circulation disorder becomes the main contradiction, and it is imperative to unblock the ventricular system as soon as possible. Therefore, for the treatment of ventricular hemorrhage, once diagnosed, blood accumulation within the ventricles should be removed as early as possible to reduce secondary brain damage, improve cerebral microcirculation, and prevent the occurrence of complications. In our group of 29 patients, the mortality rate of 17.3% (5/29) P was achieved by using measures such as extraventricular drainage, intraventricular urokinase perfusion combined with lumbar puncture to release cerebrospinal fluid.