It refers to the chronic partial or complete obstruction of the portal vein or intrahepatic branches of the portal vein, resulting in obstruction of portal blood flow and causing increased portal pressure. To reduce portal hypertension, varices may occur in the superficial abdominal wall veins located around the umbilicus after the formation of collateral circulation around the portal vein or recanalization of the obstruction. In hepatic portal hypertension, the main manifestations are portal hypertension and secondary rupture of esophagogastric fundic varices and/or associated portal hypertensive gastropathy. Patients may have recurrent vomiting of blood and tarry stools with mild to moderate splenomegaly and hypersplenism; therefore, the liver function of such patients is good, so ascites, jaundice and hepatic encephalopathy rarely occur. Occasionally, spongy degenerative collateral vessels may compress the common bile duct and varices may occur in the superficial abdominal wall veins around the umbilicus. Differential diagnosis of abdominal wall venous fury: 1. Subcutaneous venous reticular fury: It is one of the clinical symptoms of thrombophlebitis. It is a venous disease characterized by acute nonsuppurative inflammation of the venous wall and intraluminal thrombosis. Slow blood flow and vortex formation, increased blood coagulation and intimal damage are its main etiologies. Clinically, there are two categories: superficial thrombophlebitis, and deep vein thrombosis. It needs to be differentiated from venous edema, lymphedema. Limiting thrombosis and inflammation; eliminating swelling and restoring venous function as much as possible; and preventing the development of fatal pulmonary embolism are the main objectives of treatment. 2.Retinal vein varices and anger: Retinal vein obstruction is a relatively common fundus vascular disease. Its incidence is higher than that of arterial obstruction. Most cases occur in middle age or older, slightly more in men than in women, and often in one eye. The main symptom is the loss of central vision, or a part of the visual field defects, but the onset is much less acute and serious than arterial obstruction, usually still can retain part of the vision, in 3-4 months after the central vein obstruction, about 5-20% of patients can appear iris neovascularization, and secondary neovascular glaucoma. 3.Jugular vein anger: When normal person is standing or sitting, the external jugular vein is not revealed, when lying down it is slightly filled, but it is limited to 2/3 of the distance from the superior clavicle to the angle of the jaw, if it exceeds the above level or 45 degrees in semi-recumbent position, the jugular vein is filled, distended and full then it is called jugular vein anger, which indicates that the venous pressure is increased and it is abnormal. The symptoms described by the patient are not only the main clue to determine the presence or absence of jugular vein irritation, but also provide the main reference basis for the etiologic diagnosis. Long-term chronic cough with progressive dyspnea is mostly associated with right heart failure due to pulmonary heart disease; sudden onset, severe chest pain, coughing up dark red blood sputum, and dyspnea disproportionate to pulmonary signs suggest pulmonary artery embolism; irregular fever, palpitations. In cases of dyspnea and precordial pain, pericardial effusion and constrictive pericarditis should be considered after excluding other infections; in juvenile or adolescent onset, shortness of breath after exertion, weakness, palpitations, and hair groups suggest precordial disease such as primary pulmonary hypertension, pulmonary valve stenosis, Ebstein’s malformation, Eisenmenger’s syndrome, and atrial septal defect. In adolescents, palpitations and dyspnea suggest restrictive cardiomyopathy, but it is less common. Young and middle-aged patients with a history of rheumatic fever and post-activity weakness and palpitations and abdominal distention suggest rheumatic heart valve disease, such as tricuspid stenosis and/or incomplete closure.