The child, female, 11 years old, was born in Nanjing, Jiangsu Province. The child was found to have a punctate hemangioma at the inner ankle since the age of 4 years, which gradually progressed to the trunk. One year apart, black stools appeared, unrelated to diet, in moderate amounts, along with anemia. He came to the hospital on December 23, 2001 because of recurrent blood in the stool for 6 years. There was no familial history of hereditary disease. He had a clear consciousness, pale complexion and lips, a heart rate of 104 beats/min, a rhythm, strong heart sounds, and no murmurs were heard. The whole abdomen was flat and soft, no intestinal pattern and peristaltic waves were seen in the abdomen, no pressure pain and mass in the whole abdomen, and no liver and spleen were found. No polyps or other abnormalities were found on anal finger examination. There were dozens of scattered hemangiomas on the skin of the trunk and extremities, ranging in size from 1cm×1cm to 1.5cm×2cm in diameter. there were no deformities in the joints of the extremities, and the extremities moved freely. Laboratory examination: WBC 4.1×109/L, N72%, L17%, Hb86g/L, PLT281×109/L, liver and kidney function were in normal range, coagulation function was normal, gastroscopy showed: gastric body augmentation type lesion, ultrasound showed: liver, bile, pancreas and spleen did not show any occupying lesion. Diagnosis: gastrointestinal bleeding, anemia, gastrointestinal hemangioma, multiple hemangiomas of the skin. On the fourth day of admission, a fiberoptic colonoscopy was performed, which revealed seven intestinal wall hemangiomas of various sizes from the ileocecal region to the rectum. On the 14th day of admission, he underwent dissection and resection of gastric hemangioma, which revealed multiple hemangiomas scattered in the liver, 2 hemangiomas in the pancreas and large omentum, 37 hemangiomas in the small intestine and mesentery, 2 hemangiomas in the gastric body, and an ileocecal sleeve in the ileum about 100 cm from the ileocecal region. The hemangioma was removed from two parts of the gastric body, and the remaining hemangioma was not treated. Postoperative diagnosis: Bean’s syndrome. Postoperative pathology: submucosal cavernous hemangioma of the stomach. Discussion: Bean’s syndrome is also known as blue rubber bleb nevus syndrome. Bean’s syndrome is a syndrome manifested by the simultaneous appearance of spongy or capillary hemangiomas in the skin and the digestive tract, with clinical manifestations of blue rubber nevus, hemangioma, digestive bleeding and iron deficiency anemia caused by digestive bleeding. There is a risk of cerebral, pulmonary or renal embolism due to dislodged blood clots. The disease is extremely rare, with Fernandes reporting about 200 cases to date. The disease is thought to be associated with chromosome 9p aberrations, and there have been reports of brothers in the family with the disease, according to which it is thought to be autosomal dominant. There is no specific treatment for this disease, and it is generally treated conservatively with hormones or interferon. Laser or electrocautery treatment has been reported for lesions in the gastrointestinal tract, but they are prone to recurrence. Surgical removal of the bleeding lesion is generally considered when there is gastrointestinal bleeding.