We often encounter patients with gout in the clinic: they go to the hospital when their joints are red, swollen and painful, take medication for a few days, and then stop taking it until the next attack. Due to the lack of scientific treatment, the frequency of joint inflammation tends to increase and gradually affects the kidney function. To avoid these misconceptions, patients are advised to receive standardized treatment. It is currently believed that a more standardized gout treatment should be as follows: 1. Attack phase: relief of joint redness and pain with antipyretic and analgesic drugs. Uric acid-lowering therapy is usually not given during this phase, and large fluctuations in uric acid may induce aggravation of gouty arthritis attacks. (For patients who insist on taking uric acid-lowering drugs, it is advisable to maintain the usual dose of medication and not to reduce or stop it at will, so as not to cause a significant rise in uric acid) 2. Long-term control of serum uric acid at about 300umol/L is recommended to help the gradual dissolution of uric acid salts that have formed crystals, protect the kidneys and reduce attacks. Receiving standardized treatment is an effective measure to reduce inflammatory joint attacks and protect the kidneys.