Mercury toxic tremor is mainly neuromuscular tremor, mostly intentional tremor, that is, the tremor starts during the action, increases during the action, and stops after the action is completed, and the tremor increases more significantly when it is noticed by others, when it is tense, or when it is more controlled. What are the causes of mercury toxic tremor? Mercury vapor easily passes through the lipid-containing cell membranes of the alveolar wall, combines with lipids in the blood, and is quickly distributed to all tissues of the body. Mercury is oxidized to Hg2+ in red blood cells and other tissues, and accumulates by binding to proteins, making it difficult to be released again. Metallic mercury is barely absorbed in the gastrointestinal tract, only about one ten-thousandth of the ingested amount, and the absorption of mercury salts in the digestive tract is about 10%. Mercury is mainly excreted in urine and feces, and a small amount is excreted in saliva, milk and sweat, and very little is exhaled from the lungs. The half-life of elemental mercury in the body is 60 days and that of mercury salts is about 40 days, with a higher excretion in the first 4 days. Mercury ions are easily combined with sulfhydryl groups, making cytochrome oxidase, pyruvate kinase, succinate dehydrogenase, etc., which are related to sulfhydryl groups, inactive. Mercury also affects the activity of functional groups by binding to amino, carboxyl and phosphoryl groups. As the activity of these enzymes and functional groups is affected, the biological activity and normal metabolism of cells are hindered, eventually leading to cell degeneration and necrosis. In recent years, mercury has been found to damage the kidney, predominantly in the epithelial cells of the renal proximal tubule. Mercury can also cause immune dysfunction, the production of autoantibodies and the development of nephrotic syndrome or glomerulonephritis.
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