Visual loss of recognition is the loss of contact between sensory objects and previously remembered material and becoming unrecognizable, i.e., the inability to recognize. It is an acquired cognitive impairment due to localized damage to the brain. The patient is able to recognize an object through other sensory channels, but loses the ability to recognize a familiar object, self or visual space through a specific sensory channel and the corresponding senses. This inability to recognize is not due to sensory, language, intellectual, or memory deficits, nor is it due to the patient’s unfamiliarity with the object, but is often caused by damage to specific functional areas of the cerebral hemispheres. Most manifestations of anosognosia are idiosyncratic. Anosognosia, like other abnormalities of brain function, has asymmetries in both cerebral hemispheres. Visual anosognosia is a condition in which a patient with brain damage has no visual, auditory, somatosensory, consciousness or intellectual impairment, but is unable to recognize previously familiar objects by one sense but can recognize them by other senses. For example, if a patient sees a watch and does not know what it is, but touches the shape of the watch and hears the sound of the watch moving, he immediately recognizes it as a watch. The common types of visual agnosia are generalized agnosia, associative agnosia, color agnosia, and face agnosia. Patients have normal functional and structural damage to primary visual cortex area 17, lateral geniculate body, visual pathways, optic nerve and eye; focal brain damage can be in visual cortex areas 2-4 (V2, V3, V4) or inferior temporal gyrus, middle temporal gyrus, superior temporal sulcus, respectively, and also commonly in the occipital-temporal interconnection fibers.