Gouty arthritis is caused by a disorder of purine metabolism in the body, resulting in an increase in uric acid in the blood, which is deposited in the joints and forms crystals. Gout attacks are very intense, often manifesting as swelling and pain in one joint, more often in the big toe. In some severe cases, the pain can be multiple joints. If gout is not controlled for a long time, it can lead to joint destruction and the development of gout stones in the joints and kidneys. Patients usually have a history of elevated blood uric acid, and examination at the onset of swelling and pain can reveal significantly elevated blood sedimentation and C-reactive protein. Treatment is mainly divided into anti-inflammatory and pain-relieving treatment in the acute phase and uric acid-lowering treatment in the remission phase. When the joint swelling and pain attack, the inflammation should be controlled quickly, so strong anti-inflammatory and pain-relieving drugs such as etoricoxib will be used, and if the symptoms are stubborn, hormones can be added to strengthen the anti-inflammation. Patients in the acute stage should not be treated with uric acid-lowering therapy to avoid aggravating joint swelling and pain. After the pain and swelling of the joints are relieved, regular and long-term uric acid-lowering therapy should be started. The commonly used drugs are: allopurinol, benzbromarone, etc. The goal is to control the blood uric acid at about 300umol/L for a long time and stabilize it for several months, so that the previously accumulated uric acid can be excreted from the body to avoid recurrent attacks. In addition, gout patients need to pay attention to their diet, avoiding seafood, beer, thick soup, offal and other foods, eating as light as possible and drinking more water.