Many gout sufferers have experienced that after a good meal, they suddenly find that their joints are swollen and painful, and they cannot even sleep, then they rush to the hospital and ask the doctor to stop the pain. The doctor prescribes medicine for him, and the effect is very obvious. However, after a while, if he was not careful, he suddenly got pain again and had to go to the doctor again. This repeatedly makes the gout patients suffer a lot. Some patients even think that gout is an “incurable disease” and lose their confidence in treatment, casting a huge psychological shadow. In fact, the pathogenesis of gout has been well studied by the medical community, and has a complete and standardized treatment method. Gout patients can avoid the torment of repeated pain. But gout is not like a cold that can be treated in a few days, but requires perseverance to “eliminate the root cause”. Gout is caused by a metabolic disorder in the body, resulting in the production of a large number of metabolites called “purines”. Purine can be converted into uric acid and excreted in the urine. However, when uric acid rises beyond the body’s ability to excrete it, hyperuricemia develops. Long-term hyperuricemia mainly causes damage to joints and kidneys, producing gout stones in joints, kidneys, ureters and skin, etc. Long-term hyperuricemia also leads to hyperlipidemia, hyperglycemia and hypertension, which can be said to have infinite consequences. The damage of high uric acid to the body is a very long process, many people only find high blood uric acid in the examination, but do not feel any discomfort. However, as more and more uric acid accumulates in the body, the so-called “accumulation is difficult to return”, coupled with the lack of attention to diet, it will manifest itself as joint swelling and pain. Therefore, if a gout patient only stops the pain when the joints are sore, but does not completely eliminate the uric acid that has accumulated over the years, it will certainly lead to recurrent gout attacks. The treatment of gout is divided into two stages: the first stage is pain relief and anti-inflammation during acute attacks, and the second stage is uric acid lowering treatment during intermittent and chronic periods. Many patients only stay in the first stage of treatment, but in fact, the second stage of uric acid reduction is the most critical, but the most difficult to adhere to. Because this stage requires the use of uric acid-lowering drugs to control the uric acid level below 327μmol/L, the treatment time is longer, during which the dosage should be adjusted under the guidance of doctors, and the blood routine, liver and kidney function and other indicators should be reviewed regularly. Many patients are just afraid of trouble and can’t stick to it, while some think they don’t need to take medication if they are not in pain, and some are worried about the side effects of long-term medication. Therefore, as a patient, you need to understand the following two aspects: First, gout is a chronic disease, the so-called “slow disease requires slow treatment”, so you need to be mentally prepared for a long-term battle. Secondly, after understanding the pathogenesis of gout, we will understand that the acute attack of gout is only an indication, like seeing the tip of the iceberg, we should pay more attention to the huge iceberg hidden under the surface of the water, so we should not forget the pain when we are well and still need to treat it when it is not painful. Thirdly, the commonly used uric acid-lowering drugs include allopurinol, propofol, benzbromarone, etc. Their clinical use has been very standardized and mature, and the possible adverse reactions to them have been very clear. As long as the patient takes them under the guidance of the doctor and regularly checks the blood routine, liver and kidney function and other indicators, they are very safe to use. The treatment of gout needs to be persistent, and as long as the standard treatment is adhered to, gout patients can avoid the torment of repeated joint pain, as well as joint deformity, high blood sugar, high blood lipids, high blood pressure and impaired kidney function.