Thalassemia causes weak pulse mostly in infancy, manifesting as anemia, weakness, and intra-abdominal masses. Most of the thalassemia patients are born in infancy, and most of them have anemia, weakness, abdominal masses, and developmental delay. Patients with mild and intermediate types can generally live into adulthood and can participate in the workforce. If attention is paid to labor control and diet, complications can be reduced and symptoms can be improved. Diagnosis: Chinese medicine considers deficiency of endowment and weakness of kidney energy as the main causes. The kidney is the root of the innate nature, the innate kidney essence is not full. Then there is no source of biochemistry. “The child’s labor is obtained from the mother’s fetus”. It can be seen that ‘”child labor” is closely related to the parents. Kidney essence is not full at the same time also affects the function of the spleen and stomach and the growth and development of the latter, long then the blood is corrupted. This is the result of the mixed evidence of deficiency and reality. Quantitative measurement of hemoglobin is a routine clinical diagnostic method. The increase of HbA2 is the basis for the diagnosis of mild β-thalassemia. In severe β-thalassemia, HbF usually increases, sometimes up to 90%; the increase of HbA2 is usually more than 3%. In alpha-thalassemia syndrome, the percentages of HbA2 and F are usually normal, and the diagnosis often relies on the exclusion of other causes of microcytic anemia. The diagnosis of HbH disease is made when fast-moving HbH or Bart fragments are shown on hemoglobin electrophoresis. Recombinant DNA genetic mapping techniques (especially using the multiplex enzyme chain reaction method) are important in prenatal diagnosis and genetic counseling. In beta thalassemia major, the X-ray of the bones shows features of chronic bone marrow hyperactivity. The cortical layer of the skull and long bones is thinned and the bone marrow cavity is widened. The cranial laminae are distinct, and the laminae have “sun-ray” like radiolucencies in the trabeculae. Areas of osteoporosis may be present in the long bones. The conus and cranium may appear granular or frosted glassy. The finger (toe) bones lose their normal shape and are rectangular or even protruding on both sides.