Myocardial bridges are coronary arteries that travel between the myocardium and are usually congenital, and their course does not change with age.
When myocardial cells contract, they squeeze the bridged vessels, resulting in severe angina-like clinical manifestations, arrhythmias, myocardial infarction and even sudden death. Myocardial bridges cause the blood flow that should be forward during systole to flow backwards, easily damaging the endothelium of the arteries in the area, and are therefore prone to atherosclerosis. Current treatment measures include medication and surgery.
Drugs can be chosen to reduce myocardial contractility (β-receptor antagonists such as propranolol, calcium channel blockers such as nifedipine, etc.) to relieve symptoms. Separation of the wall coronary artery can also be chosen for radical treatment, but there is also a risk of recurrence.
Medications need to be applied appropriately and in accordance with medical advice.
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