The survey shows that 90% of liver cancer patients evolved from hepatitis B. The risk of patients with a history of hepatitis B is 10.7 times higher than that of other patients, and among them, the risk of liver cancer among virus carriers is 6.37 times higher than that of healthy people. In other words, most of these liver cancer patients are caused by the deterioration of hepatitis B patients, or hepatitis B virus carriers, and there is a close relationship between hepatitis and liver cancer. Why do hepatitis B patients go to liver cancer? 1. Hepatitis B virus is hepatophilic in nature, it lurks in the liver and waits for an opportunity to commit crimes against other organs of the body. The hepatophilic nature of hepatitis B virus is the initiating factor for the virus to induce liver disease, and the development of hepatitis B is closely related to the immune status of the patient’s body. The presence of the hepatitis B virus is the root cause of the persistence, fluctuation, deterioration and relapse of the disease in hepatitis B patients. 2. Hepatitis B virus genes can integrate with related genes in the nucleus of hepatocytes, and when X-gene integrates with genes in the nucleus of hepatocytes, it can lead to primary liver cancer. This is also one of the important reasons for the occurrence of hepatocellular carcinoma. 3. Hepatitis B virus mainly invades hepatocytes. When hepatocytes are infected, hepatitis B virus will use some protrusions on the shell of hepatocytes and attach to the surface of hepatocytes. When some core genes of the hepatitis B virus enter the hepatocytes, the genes of the virus can freely control the normal activities of the hepatocytes, forcing the hepatocytes to make more virus, leading to the weakening of the hepatocytes until they die. After the liver cell dies, the hepatitis B virus is released from the liver cell again and continues to infect other healthy liver cells. This process is often completed in just a few hours. After this is repeated many times, the patient’s liver will be increasingly damaged and the formerly normal liver cells will gradually be replaced by extensively fibrotic liver cells, leading to cirrhosis and even liver cancer. Daily factors that induce hepatitis B to become liver cancer 1. Alcoholism. Long-term alcohol abuse can promote the occurrence of cirrhosis or liver cancer to those with positive hepatitis B surface antigen. 2. Stale cooking oil. Stale animal and vegetable oils are toxic and may cause cancer. 3.Moldy food. People consuming moldy food can cause mold spores to invade the blood or respiratory tract and lead to physical diseases. Moldy food mostly contains aflatoxin, and long-term intake of aflatoxin can lead to cancer of the liver. 4.Leave it alone. If hepatitis B patients do not pay attention to treatment and leave it alone, hepatitis B virus will continue to infect and replicate, and inflammation will lead to liver fibrosis, which will eventually lead to cirrhosis and even liver cancer. Therefore, hepatitis B patients and family members should regularly go to the hospital for routine checkups, regular rechecking of serum transaminases, half a year to a year “two to half”, a fetal alpha-fetoprotein, and an annual ultrasound examination 5, drug abuse. The abuse of drugs is very damaging to the liver, and in the face of the increase in the variety of drugs, patients must be careful in treatment and avoid the blind use of drugs. The hepatitis B virus has a very destructive effect on the human body, and without the complete elimination of the hepatitis B virus, hepatitis B patients will never have peace. Therefore, the treatment of hepatitis B must first eliminate the hepatitis B virus, that is, to carry out the so-called antiviral treatment. In clinical practice, if chronic hepatitis B patients can undergo standardized antiviral treatment, they can maximize the inhibition or elimination of hepatitis B virus in their bodies, reduce the inflammatory necrosis and fibrosis of liver cells, delay or stop the occurrence of cirrhosis, liver cancer and their complications, thus improving their quality of life and prolonging their survival time. Liver preservation therapy and immunomodulation are only part of the comprehensive treatment of hepatitis B, and cannot replace antiviral therapy. If the hepatitis B virus replication in the patient’s body is rampant, and the patient is simply on liver-protective therapy without antiviral therapy, it is in essence a practice of putting the cart before the horse, and it is impossible to solve the problem at the root!