Gout is a kind of metabolic rheumatism, which is a group of diseases caused by long-term disorder of purine metabolism and high blood uric acid. Hyperuricemia ≠ gout Only 5-12% of patients with hyperuricemia will get gout, the rest have no symptoms, so high uric acid is not equal to gout. When blood is drawn from a patient with gout during an acute arthritis attack, about 1/3 of the patients have uric acid values within the normal range, so gout is not necessarily hyperuricemia. Most hyperuricemia can last a lifetime without symptoms, but the higher the blood uric acid level and the longer it lasts, the greater the chance of gout and urinary tract stones. That’s why it’s so important to say that hyperuricemia is closely associated with the development of acute arthritis in gout. The most common triggers for an acute attack of gout: alcoholism, high purine diet, infection, cold, exertion, drugs, trauma and surgery. Prevention of gout: 1, abstain from alcohol; 2, eat less or no food containing high purine (such as: broth, hot pot, fatty meat, animal offal, seafood, etc.) 3, drink more water, 2000-3000ml; 4, alkalinize urine (baking soda tablets or mineral water containing soda), etc. What are the causes of elevated uric acid in the organism? There are only three aspects: 1, is the intake of food containing high purine too much. It accounts for 20%. After the metabolism of food containing high purine in the body, the end product of purine – uric acid levels rise. This is called exogenous uric acid elevation. 2. The body produces too much uric acid, which is called endogenous uric acid elevation, accounting for 80%. The human body produces about 600 to 900 mg of urate daily, and 2/3 of it is excreted in the urine. 3. Excessive uric acid is excreted through the kidneys.