Gout is a crystal-associated arthropathy caused by uric acid deposition and is directly related to hyperuricemia due to disorders of purine metabolism and/or decreased uric acid excretion. The first attack mostly affects a single joint, with the first metatarsophalangeal joint being the most common, often waking up at night with severe, unbearable pain, such as cutting or burning, and localized symptoms of redness, swelling, heat and pain. If not treated in time, the episodes become more frequent and gradually develop into chronic gouty arthritis. Not only that, it is also a systemic metabolic disease, which can form gout stones and involve the kidneys, leading to kidney stones and interstitial kidney lesions to renal insufficiency. The lack of formal treatment at the beginning of the disease leads to the gradual aggravation of the disease, chronic gouty arthritis and renal insufficiency to the point of living in a wheelchair, which seriously affects the quality of life of patients. In fact, gout is a controllable disease, and the development of the disease to such a serious degree can be completely controlled through treatment. Therefore, for patients with hyperuricemia who have a history of acute gouty arthritis attacks, in addition to lifestyle interventions such as a low purine diet, drinking more water and maintaining a daily urine output of more than 2000 ml, it is recommended to go to a regular hospital for effective uric acid-lowering treatment during the interval between attacks, so that the blood uric acid can be reduced to 360 umol/L or even below 320 umol/L to control the development of the disease. The progression of the disease can be controlled.