Patients with gout should seek systematic and regular treatment at a hospital and not take drugs on their own to avoid delaying the disease and causing it to deteriorate. Recurrent attacks of acute gouty arthritis, gouty stone deposits, and chronic gouty stone arthritis can lead to joint deformities, which can cause lifelong disability in severe cases. Gouty kidney is often accompanied by proteinuria and hematuria. Even hypertension, azotemia and uremia can occur eventually with dialysis maintenance treatment. Therefore, early prevention and treatment is necessary. Patients themselves should regulate the diet and living to do the following: 1, low purine diet: purine is the source of uric acid production, reduce the consumption of food containing high nuclear protein, such food mainly livestock offal (such as heart, liver, kidney, etc.), some fish, germ, beans (will be sprouted or sprouted food, but except mung bean sprouts), gravy, broth (purine is easily soluble in water, long stewed broth contains a lot of leaching from the meat) Patients are advised to reduce the consumption of such foods under the principle of not affecting the normal nutritional intake). You can choose fresh pork, chicken, freshwater fish and milk to supplement a certain amount of protein, and fresh vegetables can be eaten more to make the diet more reasonable and help prevent gout attacks. 2, maintain the ideal weight: obesity is not only a risk factor for the development of gout, but also a factor promoting the development of gout. Gout with obesity can also affect the effect of drugs and reduce drug sensitivity. General weight loss should be about 2 to 3 weeks to lose about 2 kg of weight is appropriate. Need to pay attention to weight loss should not be too fast, because fat and other tissues if the breakdown is too fast can cause ketone bodies and lactic acid concentration increase, inhibit uric acid secretion and induce acute attacks of gout. 3, drink more water: can help uric acid excretion, daily intake of at least 2000-2500ml of water 4, avoid drinking alcohol: drinking alcohol is one of the most important triggers of gout attack. Ethanol, the main component of alcohol, can increase lactic acid in the body, which can inhibit the excretion of uric acid by the renal tubules; ethanol can also promote purine decomposition and directly raise blood uric acid; at the same time, alcohol itself can provide purine raw materials, such as beer contains a large number of purine components. Therefore, large amounts of alcohol can cause gout attacks, long-term chronic alcohol consumption can also occur hyperuricemia. Gout patients are best to quit drinking, a moment to quit should also pay attention to avoid drinking a lot of alcohol, more avoid alcohol abuse. 5, can not be strenuous exercise: strenuous exercise after the body lactic acid increased, inhibit the renal tubular excretion of uric acid and make the blood uric acid rise. It can also be due to excessive sweating, body water loss and blood volume, kidney blood flow is reduced and affect uric acid excretion, causing transient hyperuricemia. Therefore, gout patients should not be strenuous exercise. It is advisable to choose aerobic sports with low exercise intensity, such as walking, cycling, swimming, etc., while avoiding ball games, climbing, jumping and other sports intensity projects, while paying attention to rest during exercise, and should drink more water. 6, pay attention to warmth: cold causes vasoconstriction of the body surface and internal organs, including the contraction of kidney blood vessels, which causes a decrease in uric acid excretion. Cold can also induce gout attacks, so gout patients should avoid cold. 7, prevention of kidney stones: gout patients have a higher incidence of kidney stones, therefore, in order to promote uric acid excretion and prevent urinary stones, it is necessary to drink more water, while paying attention to the alkalization of urine. Patients with stones should not use drugs that promote uric acid excretion. 8, regular review of kidney function: high concentration of uric acid salt deposition in the kidney tissue can cause gouty nephropathy. Therefore, actively control hyperuricemia and regularly review renal function to prevent kidney damage and acute renal failure.