Hyperuricemia is the basis for the development of gout, and in most cases elevated blood uric acid is a precursor to gout, especially for patients with chronically elevated blood uric acid who are at high risk for a gout attack. Gout is a crystal-associated joint disease caused by urate deposition, and hyperuricemia is the basis for the development of gout. In the asymptomatic phase of gout, patients may show no discomfort and only have persistent or fluctuating elevations in blood uric acid. Many patients do not know that their blood uric acid is elevated without a blood test and continue to eat a lot of purine-rich foods, but this also increases the likelihood of a gout attack. The asymptomatic period of gout can last for years or decades and is associated with multiple factors such as gender, age, length of history of hyperuricemia, and high or low blood uric acid levels. The acute attack of gout is usually not preceded by any precursors, but often strikes at night or in the early morning, when the patient wakes up from sleep and finds that the joint is red, swollen and painful, unbearable, like a knife cut or insect bite. However, even without medication, joint symptoms may resolve on their own within a few days or 2 weeks, or even disappear. If the disease is not well controlled, acute gout attacks can occur frequently and each attack is similar, without any obvious aura. However, the absence of aura does not mean that there are no triggers. A history of high purine diet such as heavy drinking, eating seafood or animal offal within 3 to 5 days before the attack; long-term elevated blood uric acid without the use of uric acid-lowering drugs; a history of chronic gout and recent late nights and strenuous sports are all triggers for acute gout attacks. It can be seen that gout attacks are mostly sudden, but there is already elevated blood uric acid before that, which is a reminder that gout may occur.