Uric acid is the end product of purine metabolism and is mainly produced by the enzymatic breakdown of nucleic acids and other purine analogues from cellular metabolism and purines from food. The saturation and concentration of uric acid in the body at 37°C is about 420 μmol/L (7 mg/dl), above which hyperuricemia is defined as hyperuricemia. Most patients with primary hyperuricemia have no clinical symptoms and often have manifestations of metabolic syndrome. Patients with chronic hyperuricemia may lead to gout and therefore exhibit symptoms of gout. So the common symptoms of hyperuricemia include: 1. Asymptomatic phase: only fluctuating or persistent hyperuricemia, the time from the increase of blood uric acid to the appearance of symptoms can be years to decades, and some can remain symptom-free for life, but the prevalence of gout increases with age. 2, gouty arthritis: often first in the first metatarsophalangeal joint, or ankle, knee joints, the onset of the disease is rapid, the first attack often involves a single joint, can be spontaneous remission, repeated attacks will gradually increase the number of joints involved. Gout stones: In patients with untreated first symptoms, gout stones may appear in about 70% of patients after many years, often in the first metatarsophalangeal joint, auricle, forearm extension, finger joints, elbow joints and other parts. 4, renal lesions: mainly manifested in both gouty nephropathy and uric acid kidney stones. 5. Eye lesions: gout stones, recurrent conjunctivitis, keratitis and sclerositis may occur. Therefore, patients with hyperuricemia do not necessarily have clinical symptoms, but if the above-mentioned symptoms appear, the diagnosis should be further confirmed with the patient’s medical history and relevant examinations.