Facial palsy, which refers to facial muscle paralysis, is a condition caused by damage to the facial nerve from various causes. Depending on the site of nerve damage, facial palsy should be divided into peripheral facial palsy (peripheral facial nerve palsy) and central facial palsy (central facial nerve palsy). The specific clinical manifestations of the two are different: peripheral facial palsy refers to facial palsy caused by lesions in the motor fibers of the facial nerve. The lesion may be located below the facial nerve nucleus, such as the inferior pontine brain, facial nerve canal, middle ear or parotid gland. All the expression muscles on the side of the lesion are paralyzed. It is characterized by the inability to close the eyelids, frown, cheek puffing, etc. There may be auditory changes, loss of taste in the first 2/3 of the tongue and salivary secretion disorders. Central facial palsy refers to facial palsy caused when the lesion is located between the nucleus accumbens and the central cerebral cortex, i.e., when one side of the cortical brainstem bundle is damaged. The cells in the upper part of the facial nucleus receive fibers from the cortical brainstem bundle on both sides, and their axons form the motor fibers of the facial nerve, which innervate the expression muscles above the ipsilateral eye fissure, while the cells in the lower part of the facial nucleus only receive fibers from the contralateral cortical brainstem bundle, and their axons form the motor fibers of the facial nerve, which innervate the expression muscles below the ipsilateral eye fissure, so central facial palsy manifests as paralysis of the facial expression muscles below the eye fissure on the opposite side of the lesion, often accompanied by Therefore, central facial palsy is characterized by paralysis of the facial expression muscles below the eye fissure on the opposite side of the lesion, often accompanied by paralysis of the limbs on the same side as the facial palsy, and the absence of taste and salivation.