I. Etiology of hepatic hemangioma The etiology of hepatic hemangioma is not yet clear, and is mostly thought to be related to congenital developmental abnormalities, which may be related to the following factors. (1) deformation of capillary tissue after infection, resulting in capillary dilation; (2) vascular dilation and formation of vacuoles after local necrosis of liver tissue, and vascular congestion, dilation, and finally formation of vacuoles around necrotic liver tissue; (3) regional blood circulation stagnation in the liver, resulting in vascular spongy dilation, and persistent venous blood stasis in the liver, resulting in venous expansion; (4) intrahepatic hemorrhage, hematoma mechanization, and vascular recanalization, resulting in vascular dilation; (5) The most acceptable theory is that the abnormal development of blood vessels causes spongy dilatation of blood vessels. Types of hepatic hemangioma ①Hepatic cavernous hemangioma: the most common. ②Sclerosing hemangioma: ③Hepatic capillary hemangioma: rare. ④Hemangiopericytoma: rare, intermediate between benign hemangioma and hepatic hemangioendothelial cell sarcoma. Clinical manifestations of hepatic hemangioma The clinical manifestations of hepatic cavernous hemangioma are related to the location, size, growth rate and the degree of liver parenchymal involvement of the tumor. Indications for interventional treatment of hepatic hemangioma The main interventional treatment for hepatic hemangioma is hepatic artery embolization (HAE). Indications for hepatic artery embolization for hepatic hemangioma: hepatic hemangioma larger than 5cm, regardless of the location, scope and number, is currently mostly used for the treatment of hepatic hemangioma that cannot be surgically removed. V. Advantages of hepatic artery embolization Hepatic artery embolization is an effective method for the treatment of hepatic hemangioma, with the advantages of wide indications, little damage, fast recovery and good efficacy. After hepatic arteriography, the catheter is super-selected and inserted into the target vessel for embolization treatment according to the image. If it can cross the gallbladder artery, anhydrous alcohol or sodium cod liver oil acid can be used, and then gelatin sponge can be used to strengthen the embolization, otherwise, it can be used carefully according to the tumor vascular condition. If it fails to cross the gastroduodenal artery, it is safer to use iodized oil plus gelatin sponge strips. VII. Evaluation of efficacy of interventional treatment Hepatic hemangioma is mainly composed of blood-filled, dilated blood sinusoids. The terminal embolic agent stays in these sinusoids to form thrombus, thrombus mechanization and fibrosis can transform the tumor into fibroid structure, anhydrous alcohol can also cause destruction of endothelial cells of blood sinusoids and permanent occlusion of blood sinusoids. In this way, the tumor will shrink and will not rupture and bleed, achieving a therapeutic effect, and some patients can heal completely with satisfactory results. The etiology of hepatic hemangioma is not yet clear, and is mostly thought to be related to congenital developmental abnormalities, which may be associated with the following factors (1) deformation of capillary tissue after infection and capillary dilation; (2) vascular dilation forming vacuolation after local necrosis of liver tissue, and vascular congestion, dilation, and finally vacuolation around necrotic liver tissue; (3) regional blood circulation stagnation in the liver leading to vascular formation of spongy dilation, and persistent venous blood stasis in the liver leading to venous expansion; (4) vascular dilation after intrahepatic hemorrhage, hematoma mechanization, and vascular recanalization; (5) The most acceptable theory is that the vasculature is spongy and dilated due to abnormal development of blood vessels.