Hyperuricemia is a frequently encountered problem in clinical laboratory tests, but it is not synonymous with gout. Most hyperuricemia does not attack gout for life, and 5-12% of patients with hyperuricemia develop gout. It is now recognized that hyperuricemia at a certain high value will form urate crystals that are deposited in tissues and joint cavities causing gout. However, some people with chronic hyperuricemia do not develop gout does it require treatment? It is usually thought that treatment is not necessary, but persistent hyperuricemia may cause urate crystals to be deposited in the renal pelvis, ureter or in the renal tubules and interstitium, causing kidney damage and kidney stones. Therefore, we should look for the causes of high uric acid, such as drug factors (diuretics, antihypertensive drugs, chemotherapy drugs), diet (high purine diet, alcoholism), certain diseases (kidney disease, blood disease, diabetes) or obesity, so we should avoid triggering factors such as obesity, high purine and high calorie diet, alcoholism, overwork, trauma, wet and cold, and mental stress. People with high uric acid should have regular checkups or consider uric acid-lowering medication if they have the following conditions: 1. clinical symptoms of gout; 2. clear family history of gout and urinary stones; 3. 24-hour uric acid excretion greater than 1000 mg; 4. controlled by diet or discontinued medication affecting uric acid metabolism, and the blood uric acid value thrown for 6 months is greater than 9 mg|dl.