I. Objectives Portal hypertension is characterized by four syndromes: rupture and bleeding of the esophagogastric fundal varices, splenomegaly with hypersplenism, portal hypertensive gastropathy and ascites. The first three syndromes require surgical intervention, while ascites can be reduced or even eliminated by surgically lowering the portal venous pressure. At the same time, any surgical method should not deprive the portal vein too much of hepatic perfusion to prevent hepatic decompensation or even failure. Small-caliber splenic shunt combined with flow-breaking surgery is designed for the above objectives. Characteristics 1. Anatomically, the inferior vena cava has a shallow location, thick vessel wall, no vascular variation, easy to anastomose with the splenic vein, and the surgical operation is easy to be completed; the pressure difference between the inferior vena cava and the splenic vein is large, the blood flow is fast, and the anastomosis is not easy to be embolized. 2, The caliber of venous anastomosis is between 0.6-0.8cm, which can effectively reduce the pressure of portal vein but not divert the blood from portal vein too much, so as to maintain the reserve function of liver and slow down the time of hepatic decompensation. 3.Splenic vein is against hepatic blood flow, and the shunt is mainly venous blood from gastrosplenic hypertension area, which does not affect the blood from superior mesenteric vein, which is rich in “hepatic nutrient factor”, to enter the liver. According to the portal vein pressure, the size of anastomosis can be controlled so that the portal vein pressure is slightly higher than 30cmH2O, which can not only prevent rebleeding caused by the re-formation of collateral circulation when the pressure is too high, but also prevent the occurrence of hepatic encephalopathy caused by insufficient hepatic perfusion when the pressure is too low. Postoperative rebleeding rate, ascites disappearance rate and gastric disease improvement rate were significantly lower than that of the patients who underwent the same period of flow-cutting surgery, while the incidence of hepatic encephalopathy was similar to that of flow-cutting surgery, and all of them were mild cases, which were rapidly improved by dietary regulation and drug treatment. The rate of postoperative anastomotic thrombosis in patients was extremely low, so it would not lead to the failure of shunt surgery and could effectively reduce the portal pressure in the long term.