Hyperuricemia is a metabolic disease caused by impaired purine metabolism. It is clinically classified into two main categories: primary and secondary. In general, uric acid increases with age, and is particularly pronounced in women after menopause. The level of blood uric acid is influenced by multiple factors such as race, dietary habits, region, age, and body surface area. The saturation and concentration of uric acid in the body at 37°C is about 420 μmol/L (7 mg/dl) in men and 357 μmol/L in women, and above this concentration is considered hyperuricemia, and long-term hyperuricemia may lead to gout. The treatment of this disease is not related to age mainly includes the following aspects: Treatment principles for primary hyperuricemia: control hyperuricemia and prevent urate deposition; rapidly terminate acute arthritis attacks; prevent uric acid stone formation and renal function damage. 1.General treatment: control the total calorie of diet; limit the consumption of alcohol and high purine food; drink a lot of water (about 2000ml/day) to increase the excretion of uric acid; use drugs that inhibit uric acid excretion such as thiazide diuretics with caution; avoid triggering factors and actively treat related diseases. 2, treatment of hyperuricemia: mainly includes The first category: uric acid excreting drugs, this drug mainly through the inhibition of proximal renal tubular reabsorption of uric acid salt, so as to increase the excretion of uric acid and reduce the level of uric acid, suitable for people with good kidney function. For example, benzbromarone and propofol. The second category: drugs that inhibit uric acid production, through the inhibition of xanthine oxidase, is the reduction of uric acid production, applied to excessive uric acid production or unsuitable for the use of uric acid excretory drugs, mainly allopurinol and febuxostat. The third category: alkaline drugs, sodium bicarbonate can alkalize the urine, so that uric acid is less likely to accumulate in the urine to form crystals. 3, treatment of acute gouty arthritis: after hyperuricemia develops into gout, colchicine, non-steroidal anti-inflammatory drugs and glucocorticoids are the first-line drugs for the treatment of acute gouty arthritis and should be used early. Management of interictal and chronic phases of gout attacks: For patients with frequent attacks of acute gouty arthritis, chronic gouty arthritis or gout stones, uric acid-lowering therapy should be adhered to. Treatment principles for secondary hyperuricemia: actively treat the primary disease; try to avoid or reduce the use of drugs and methods that may trigger and/or aggravate hyperuricemia; control the acute gouty arthritis attack as soon as possible. In addition, hyperuricemia and gout are often associated with metabolic syndrome, so comprehensive treatment such as hypotension, lipid reduction, weight loss and improvement of insulin resistance should be actively carried out. Therefore, the treatment of hyperuric acid has nothing to do with age, and the specific treatment method should be chosen under the guidance of clinicians according to the cause and condition of the patient.