1.Calorie and carbohydrate Supply of sufficient calories to meet the metabolism of brain tissue, reduce tissue protein decomposition in the body and promote the recovery of liver function is a necessary part of the treatment process of hepatic encephalopathy to ensure. (1) When the patient is in coma, provide 1200~1400kcal of calories per day, completely provided by glucose, which can be input by intravenous or nasal feeding, and stop using protein. (2) After the patient is resuscitated, the calories can be slightly increased to 1200~2000kcal with the improvement of his condition, and the protein supply is 20~30g/d. Carbohydrates still account for 70%~75% of the calorie proportion, and the remaining small portion can be provided by fat. 2.Protein For patients with hepatic encephalopathy, reduce the source of ammonia. Inhibiting its production and reducing its absorption is the focus of treatment. Controlling the intake of protein in the diet is one of the basic measures to prevent the elevation of blood ammonia. (1) Protein must be discontinued or reduced to trace amounts in comatose patients, and glucose is used to maintain nutrition. However, stopping protein for too long will lead to the formation of endogenous ammonia from proteolysis in the body, which will affect the repair and regeneration of liver cells and aggravate swelling and ascites. (2) After the patient is resuscitated, daily protein containing 20-30g can be fed orally or intranasally. If the patient’s symptoms are not progressively aggravated, blood ammonia is not elevated, liver function is not deteriorated, and the mental condition gradually improves, the protein in the diet can be increased by 10g/d for 2-3 days until it reaches 50g/d. (3) Food containing less ammonia should be used. (3) Fat The use of fat should be in small doses, 1g/d daily is more effective. Medium-chain fatty acids plus essential amino acids should be used as much as possible, supplying no more than 40% of the total calories. For coma patients should try to use fat carefully. 4, water, electrolytes, vitamins liver failure, the intake of various vitamins and produce absorption disorders, poor utilization, resulting in increased vitamin loss and storage depletion, and the application of large amounts of glucose also increases the consumption of vitamins. The vitamins known to be related to liver function (B1, B2, B6, B12, C, A, E, K, folic acid, pantothenic acid, biotin, nixic acid) must be supplemented comprehensively and in doses several times higher than the physiological amount, preferably in combination, so as not to affect the balance between vitamins.