Hyperuricemia is the basis for the development of gout, which can manifest without any discomfort, so gout attacks are usually unpredictable. Gout is a crystal-associated arthropathy caused by uric acid deposits, and hyperuricemia is the basis for gout. In the asymptomatic phase of gout, patients may have no discomfort and only a persistent or fluctuating increase in blood uric acid. Many patients do not know that their blood uric acid is elevated without a blood test and continue to eat a lot of purine-rich foods, but this also increases the likelihood of a gout attack. The asymptomatic period of gout can last for years or decades and is associated with multiple factors such as gender, age, length of history of hyperuricemia, and high or low blood uric acid levels. The acute attack of gout is usually not preceded by any precursors, but often occurs at night or in the early morning, when the patient wakes up from sleep and finds the joint red, swollen and painful, unbearable, like a knife cut or insect bite. However, even without medication, joint symptoms may resolve on their own within a few days or 2 weeks, or even disappear. If the disease is not well controlled, acute gout attacks can occur frequently, with each attack being similar and without obvious aura. It should be noted that the absence of aura is not the same as the absence of triggers. Most patients have a history of drinking a lot of alcohol, eating a high purine diet such as seafood or animal offal within 3 to 5 days before the attack, in addition to long-term elevated blood uric acid without the use of uric acid-lowering drugs; chronic gout history of recent late nights, strenuous sports, etc. are all triggers for acute gout attacks. This shows that although there is no aura of acute gout attack, there are triggering factors, and controlling the triggering factors can also reduce the frequency of acute gout attack.