The cause of the disease is not yet clear, but is generally considered to be related to chronic urinary tract infections, obstruction, stones, neurogenic diseases, chemical toxic stimulation, foreign body stimulation, hormonal imbalance in the body, anatomical abnormalities of the urinary tract, and mental and emotional factors. The development of adenoid cystitis is a gradual process: simple hyperplasia of the migrating epithelium → Brunn’s buds → Brunn’s nest → cystic cystitis → adenoid cystitis. Clinical symptoms: The main clinical symptoms are O A B symptoms such as urinary frequency, urinary urgency, increased nocturia, hematuria, or lower urinary tract symptoms (1ow er urinary tract sym ptom s, L U T S) such as a sense of incomplete urination and straining to urinate. Among them: 71% of urinary frequency, 45% of painful lower abdominal perineal pain, 43% of difficulty in urination, and 36% of carnal hematuria. Of these, 79% had microscopic hematuria (≥+) and 67% had urinary tract infection (WBC ≥+). In patients whose urinary leukocytes disappeared after anti-infective treatment and whose microscopic hematuria and urinary frequency still persisted, early adenocystitis was often detected by cystoscopy and biopsy. The disease is not characteristic but requires attention. The cystoscopic classification of adenoid cystitis is: papillomatous; follicular or villous edema; chronic inflammation; and no significant mucosal changes. The predominance of the triangle and bladder neck in this group may be related to the susceptibility of this region to recurrent irritation by chronic infection or to its anatomic specificity. Adenoid cystitis is a precancerous lesion and should be closely observed and actively treated. Early stage adenocystitis: remove the cause, observe appropriately (microscopy every 1-2 years), and treat symptomatically. Late stage adenocystitis: removal of the cause, close observation (microscopy every 6 months-1 year), electrodesiccation, electrocautery, total cystectomy.