Adenoid cystitis is a rare clinical glandular hyperplasia-like lesion, the etiology of which is unclear and may be due to the development of mucosal epithelial hyperplasia and embryonic remnants of the bladder, mostly associated with urinary tract infections, but also scholars believe that it is the result of the chemotactic changes in the normal bladder urothelium caused by chronic stimulation of bladder infection, obstruction, and stones. After chronic stimulation of the bladder epithelium the basal cells show focal hyperplasia forming nests of cells and growing towards the lamina propria called Brunn’s nest, which in turn degenerates in the center of the nest and forms a cystic cavity for cystic cystitis and finally the formation of columnar epithelium in the cavity, which is known as adenoid cystitis. There are many factors associated with its transformation. However, it has also been suggested that in recent years there has been a marked increase in reports of adenocystitis, which is inconsistent with the incidence of adenocarcinoma of the bladder. Adenoid cystitis occurs in the bladder triangle, around the bladder neck and the ureteral opening and can be classified according to the morphology of the lesion as follows: 1. follicular edema type, which is manifested as a lamellar infiltrative follicular edema bulge or villi-like hyperplasia, and is clinically common; 2. papilloma-like type, which is manifested as papillae with tips, mucosal congestion, edema, and is easily misdiagnosed as papilloma; 3. chronic inflammatory type, which is manifested as localized The mucosa is rough and the vascular texture is increased; 4. The mucosa is not significantly altered, the mucosa is generally normal and is found on random biopsy, and this type is easily missed. The clinical manifestations of this disease are mainly bladder irritation symptoms such as urinary frequency, urgency, painful urination and painless carnal hematuria or microscopic hematuria.Manco et al. reported that intracavitary ultrasound can be used for the diagnosis of adenoid cystitis.Ultrasound examination shows bladder wall thickening or intravesical occupying lesions with a detection rate of about 80%. Xiao Yajun et al. concluded that hydrogen peroxide cystography helps to differentiate adenoid cystitis from bladder tumors, and despite these manifestations, the diagnosis is confirmed mainly by cystoscopy plus biopsy. There are many treatment options for adenoid cystitis, including transurethral electrodesiccation, transurethral laser cautery and sequential bladder perfusion with anticancer drugs. The first step should be to eliminate chronic irritants such as infection, obstruction and stones, and anti-infective therapy alone is often not effective. Patients with adenocystitis confirmed by pathological examination should be actively treated with electrodesiccation or electrocautery to remove the lesions, and postoperative treatment with mitotoxin, atropine and hydroxycamptothecin by regular intravesical perfusion, which can remove residual lesions and prevent recurrence and malignant transformation. In our group of 36 patients treated with transurethral electrocautery plus intravesical drug infusion, 15 of them had no recurrence with satisfactory results and no serious complications. Adenoid cystitis is prone to recurrence, and the use of transurethral electrocautery is mildly invasive to patients and can be repeatedly performed, and can be the preferred surgical treatment for adenoid cystitis.