Kawasaki disease is a vasculitis syndrome, also known as cutaneous mucosal lymph node syndrome. The greatest danger of Kawasaki disease is damage to the coronary arteries, which is the leading cause of coronary artery disease in children and a potential risk factor for coronary heart disease in adulthood. Kawasaki disease, also known as mucocutaneous lymphadenopathy syndrome, invades small and medium-sized blood vessels throughout the body causing vasculitic lesions. Invasion of the cardiovascular system is often the leading cause of death in patients with Kawasaki disease. In the acute phase, it may cause myocarditis and pericarditis, leading to heart failure or arrhythmia. Coronary artery aneurysms may develop in 15-20% of patients with Kawasaki disease at 1 to 3 weeks of illness (average of about 10 days). More than 50% of coronary aneurysms disappear within 1 to 2 years, especially the common small to medium-sized coronary aneurysms less than 8 mm in diameter. As for large coronary aneurysms over 8mm in diameter, they are often not completely eliminated by follow-up and can easily form thrombi resulting in acute myocardial infarction or coronary aneurysm rupture, both of which may cause sudden death (sudden death rate is about 2% of all patients). Myocardial infarction usually occurs within 6 to 8 weeks of onset. Myocardial hypoxia may also occur later due to narrowing or calcification of the coronary arteries caused by scar tissue after the dilatation of the coronary arteries has healed.