Myofasciitis, also known as “low back muscle injury”, “low back fibrositis”, “low back fascial pain syndrome”, etc., refers to the aseptic inflammatory reaction of muscles and fascia. The acute onset of myofasciitis can be triggered when the body is stimulated by wind and cold, fatigue, trauma or improper sleep position, etc. Acute or chronic injury or strain to the muscles, ligaments and joint capsule of the shoulder, neck and lumbar region is the basic cause of this disease. As a result of not being treated thoroughly during the acute phase and becoming chronic, or due to repeated strain, wind and cold, the patient may experience recurrent chronic muscle pain, weakness and weakness. Pathophysiology Muscles and ligaments are the power base of various activities of the human body, and their end devices are the skeletal attachments of their muscles, which are the power transmission hubs that drive bones and joints, and are also the parts where stresses are concentrated and intersected, so they are very easy to be injured. Repeated injury to the local muscle, when the injury healed can leave scars or adhesions, scar tissue can reduce the number of local blood vessels or small diameter, the occurrence of local microcirculation blood flow regulation ability to reduce the muscle blood supply and anaerobic work ability to lose. The local ischemia of the muscles causes pain due to stimulation of the peripheral nerves, and the patient cannot tolerate prolonged physical activity or even sedentary activities. For example, long-term incorrect posture or psychological depression can cause physiological contracture at the level of local muscle segments, long-term repeated muscle spasm causes muscle ischemia, sterile exudation, scar formation, local myofascia often undergoes postural load and extreme tension, fatigue causes postural injury, repeated exertion causes micro-tearing muscle damage, and pain-causing substances appear around the microvascular reaction zone of myofascia. Rheumatoid myositis, ankylosing spondylitis or viral myositis cause peripheral nerve compression due to muscle swelling, which may form nociceptive sensitive points or painful myocardial sclerosis for a long time and undergo a complex intertwined reaction process of long-term local irritation, inflammation, healing, hyperplasia or scarring, with painful local tissues and calcification of inflammatory exudate deposits and development of myoclonus. Hypoxia or lack of energy metabolism may be secondary to a decrease in local blood flow and is an important mechanism of pain in CMPS, which can cause muscle dysfunction and tissue destruction, so any means of improving microcirculation to the muscles and nerves even local massage or walking can provide some relief from the painful symptoms of CMPS.