Human beings use male and female sex to allow sperm and eggs to “meet” in order to fulfill their mission of conceiving and reproducing new life. After intercourse, the rich seminal plasma, which is surrounded by a large number of sperm, enters the female reproductive tract. Semen exists in the male reproductive tract in a liquid state, but after ejaculation from the mouth of the penis, a coagulation factor produced by the seminal vesicle gland comes into play, causing semen to coagulate and turn into a jelly-like milky substance, which on the one hand prevents semen from flowing out of the female reproductive tract, and on the other hand allows sperm to get sufficient rest and energy. Of course, in the state of semen coagulation, sperm motility is also significantly limited. After about 10 minutes, another liquefaction factor produced by the prostate gland in the seminal plasma then takes effect, automatically liquefying the seminal plasma into a thin, flowing liquid. As a result, the sperm in the reproductive tract is no longer sticky and coagulated, and at this point it gains enough energy to penetrate the cervical mucus and advance deeper into the reproductive tract; eventually it may reach the fallopian tube to meet the egg. This shows that in case the normal liquefaction of semen does not occur, the sperm will lose its normal activity. The coagulation-liquefaction process of semen is closely related to the catalysis of enzymes. A protein-like substance secreted by the seminal vesicle gland forms the matrix of the “coagulation factor”, while a protein hydrolase secreted by the prostate gland is involved in the liquefaction process. If these accessory glands are infected or otherwise diseased, this can lead to dysregulation of the enzyme system. When the prostate gland is inflamed, the lack of protein hydrolase makes the semen of some patients not liquefy or liquefy well, and the viscosity is too high, resulting in infertility. The incidence of male infertility due to semen non-liquefaction is generally about 7% to 9%, while semen non-liquefaction accounts for 30% to 40% of the infertility caused by male semen abnormalities. The most important “killer” is prostatitis. The most important reason for this is that 90% of the patients with seminal fluid are suffering from prostatitis, and 12% of the patients with prostatitis have seminal fluid that is not liquefied. The coagulation protein is mainly from the seminal vesicle gland, while the liquefaction factor is mainly from the prostate gland. The enzymes as liquefaction factors become less active when the prostate secretion activity decreases, or the balance between liquefaction and coagulation factors is disturbed by the increase of a substance called prostatic enzyme secreted by the seminal vesicle gland, which manifests as semen non-liquefaction or delayed semen liquefaction. The first step is to actively treat the primary pathology such as prostatitis and restore its normal function. The semen can only liquefy normally when the enzyme secretion function is normal. There are some patients with prostatitis who do not have liquefaction abnormalities. The possible explanation is that these patients may be combined with seminal vesiculitis, i.e., they also have reduced secretion activity of the seminal vesicle gland, which causes the coagulation and liquefaction to reach a new balance at a reduced level. Treatment: The first step is to actively treat prostatitis and restore the normal function of the period. The enzyme secretion function is normal for the semen to liquefy normally. If it is bacterial prostatitis, it is advisable to use effective antibiotics for regular treatment, it is best to perform bacterial culture of the prostate fluid and use antibiotics according to the culture and drug sensitivity results. The actual fact is that you can find a lot of people who are not able to get a good deal on a lot of things. The impact on semen non-liquefaction is second only to prostate disease, varicose sperm can not only cause male infertility through various mechanisms, but also because varicose sperm makes the pelvic cavity obviously congested, resulting in the disruption of the function of the reproductive system, including the prostate gland, thus reducing the liquefaction factor secreted by the prostate gland, plus some patients due to varicose sperm caused by testicular atrophy, so that the testicular endocrine function The combined effect of these two factors leads to the occurrence and development of semen non-liquefaction. Studies have concluded that the incidence of semen opacity in combination with varicocele is one times higher than that of the general population without varicocele. Treatment: Generally speaking, first degree varicocele does not require treatment, while treatment of second or third degree varicocele is mainly surgical. Varicocele ligation is by far the most commonly performed procedure. The body itself may also have an “inside job”. The effect on semen opacity can also be due to testicular pathology, which is mainly characterized by small (